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A cut is any partition of the vertices into two groups, S and V S What this property says is that it is always safe to add the lightest edge across any cut (that is, between a vertex in S and one in V S), provided X has no edges across the cut Let s see why this holds Edges X are part of some MST T ; if the new edge e also happens to be part of T , then there is nothing to prove So assume e is not in T We will construct a different MST T containing X {e} by altering T slightly, changing just one of its edges Add edge e to T Since T is connected, it already has a path between the endpoints of e, so adding e creates a cycle This cycle must also have some other edge e across the cut (S, V S) (Figure 83) If we now remove this edge, we are left with T = T {e} {e }, which we will show to be a tree T is connected by Property 1, since e is a cycle edge And it has the same number of edges as T ; so by Properties 2 and 3, it is also a tree Moreover, T is a minimum spanning tree Compare its weight to that of T : Both e and e cross between S and V S, and e is speci cally the lightest edge of this type Therefore w(e) w(e ), and weight(T ) weight(T ) Since T is an MST, it must be the case that weight(T ) = weight(T ) and that T is also an MST Figure 53 shows an example of the cut property Which edge is e weight(T ) = weight(T ) + w(e) w(e ).

niques are quite sensitive if performed carefully, but we are chastened by the article from Pandit and colleagues, who found falsenegative tests in up to 42 percent of examinations They found that testing of the central eld with a 5-mm red target was less likely to give misleading results when compared to the standard of automated perimetry Similarly, a central scotoma may be identi ed by having the patient xate with one eye on the examiner s nose, on which the examiner places the index nger of one hand or a white-headed pin and has the patient compare it for brightness, clarity, and color with a nger or pin held in the periphery If any defect is found or suspected by confrontational testing, the elds should be charted and scotomas outlined on a tangent screen or perimeter Highly accurate computer-assisted perimetry is now available in most ophthalmology clinics Although the commonly used automated techniques encompass only the central visual eld, this is more than adequate to detect most clinically important defects The method of testing by double simultaneous stimulation may elicit defects in the central processing of vision that are undetected by conventional perimetry Movement of one nger in all parts of each temporal eld may disclose no abnormality, but if movement is simultaneous in analogous parts of both temporal elds, the patient with a parietal lobe lesion, especially on the right, may see only the one in the normal right hemi eld In young children or uncooperative patients, the integrity of the elds may be roughly estimated by observing whether the patient is attracted to objects in the peripheral eld or blinks in response to sudden threatening gestures in half of the visual eld A common abnormality disclosed by visual eld examination is concentric constriction This may be due to severe papilledema, in which case it is usually accompanied by an enlargement of the blind spot A progressive constriction of the visual elds, at rst unilateral and later bilateral, associated with pallor of the optic discs (optic atrophy), should suggest a chronic meningeal process involving the optic nerves (syphilis, cryptococcosis, sarcoid, lymphoma) Long-standing, untreated glaucoma and retinitis pigmentosa are other causes of concentric constriction Marked constriction of the visual elds of unvarying degree, regardless of the distance of the visual stimulus from the eye ( gun-barrel or tunnel vision), is a sign of hysteria With organic disease, the constricted visual eld naturally enlarges as the distance between the patient and the test object increases A regional constriction of the eld, especially in a nasal quadrant, usually signi es early optic atrophy, as mentioned earlier; it is the rst sign that chronic papilledema is threatening the patient s vision.

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Anatomic diagnosis By history Elicitation of clinical facts By neurologic examination Interpretation of symptoms and signs in terms of physiology and anatomy Syndromic formulation and localization of the lesion Mode of onset and course Other medical data Appropriate lab tests (Anatomic diagnosis) I Figure 1-1 Steps in the diagnosis of neurologic disease II III (Pathologic or etiologic diagnosis) IV

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We are ready to justify Kruskal s algorithm At any given moment, the edges it has already chosen form a partial solution, a collection of connected components each of which has a tree 132

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